Brain fog often originates from an alteration of the intestinal barrier, where inflammatory compounds such as LPS enter the circulation, causing neuroinflammation and cognitive decline.

noli's read

The concept of the gut-brain axis is solidly established by scientific research, notably through observational studies and animal models that confirm constant bidirectional communication. The idea that lipopolysaccharides (LPS) can induce systemic inflammation is documented, although the direct causal link in humans for 'brain fog' remains an active area of research rather than a settled clinical consensus. The notion of intestinal permeability (leaky gut) is often used in the wellness community to describe this phenomenon, although this term is sometimes debated in conventional medical literature due to the complexity of barrier mechanisms. Stress and cortisol are indeed recognized in numerous studies for influencing intestinal motility and permeability, creating notable feedback loops. It is therefore scientifically plausible that dysbiosis or intestinal inflammation impacts cognition, but one should remain cautious regarding the fact that brain fog is a multifactorial symptom that does not depend solely on the gut.

the sources

No named source held this one up — that absence is part of the verdict.